Changing the strength of connections between neurons is widely assumed to be the mechanism by which memory traces are encoded and stored in the central nervous system.
In its most general form, the synaptic plasticity and memory hypothesis states that "activity-dependent synaptic plasticity is induced at appropriate synapses during memory formation and is both necessary and sufficient for the information storage underlying the type of memory mediated by the brain area in which that plasticity is observed.
Changing the strength of connections between neurons is widely assumed to be the mechanism by which memory traces are encoded and stored in the central nervous system. In its most general form, the synaptic plasticity and memory hypothesis states that "activity-dependent synaptic plasticity is induced at appropriate synapses during memory formation and is both necessary and sufficient for the information storage underlying the type of memory mediated by the brain area in which that plasticity is observed.
In these cases, there is an elegant isomorphism - isomorphism is a map that preserves sets and relations among elements - between the physiological change and the behavioural change that has been likened to a ‘cellular alphabet of learning
if you remember an event you will automatically remember the context where it happened. Realizing this kind of association requires distributed temporo-spatial representations of events and contexts that can be discriminated (pattern separation) and sometimes generalized (pattern completion), with multiple events and contexts overlaid in a distributed manner within a common neural circuit
source; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3843897/
In its most general form, the synaptic plasticity and memory hypothesis states that "activity-dependent synaptic plasticity is induced at appropriate synapses during memory formation and is both necessary and sufficient for the information storage underlying the type of memory mediated by the brain area in which that plasticity is observed.
Changing the strength of connections between neurons is widely assumed to be the mechanism by which memory traces are encoded and stored in the central nervous system. In its most general form, the synaptic plasticity and memory hypothesis states that "activity-dependent synaptic plasticity is induced at appropriate synapses during memory formation and is both necessary and sufficient for the information storage underlying the type of memory mediated by the brain area in which that plasticity is observed.
In these cases, there is an elegant isomorphism - isomorphism is a map that preserves sets and relations among elements - between the physiological change and the behavioural change that has been likened to a ‘cellular alphabet of learning
if you remember an event you will automatically remember the context where it happened. Realizing this kind of association requires distributed temporo-spatial representations of events and contexts that can be discriminated (pattern separation) and sometimes generalized (pattern completion), with multiple events and contexts overlaid in a distributed manner within a common neural circuit
source; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3843897/
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